Peptic ulcer, Peptic ulcer signs, Peptic ulcer Herbal Cure, Peptic ulcer Herbal treatments, Peptic ulcer Symptoms



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Peptic ulcer Herbal Cure and Herbal Treatment Information


Ayurvedic and herbal product
s belong to the Alternative methods of treating any disease.


 
This form of treatment is particularly popular and being widely practiced in India. Herbal treatment in India is called "Ayurvedic" treatment. It is Practiced in India for over 5,000 years and a recognized system of traditional medicine by World Health Organization (WHO).

Can ayurvedic or herbal treatments Cure Peptic Ulcer?
Because Ayurveda believes that Human Body positively respond to the natural healing which includes natural remedies. It might take some long time(with compare to Allopathic methods) to achieve full cure from Peptic Ulcer with help of Herbal medicines, but when you are fully cured then it would last throughout your life. Herbal medicines dosn't make you dependent and the best part is that There is almost NO side effect in most of the herbal products.

What Is Peptic Ulcer?

A peptic ulcer, also known as PUD or peptic ulcer disease is an ulcer (defined as mucosal erosions equal to or greater than 0.5 cm) of an area of the gastrointestinal tract that is usually acidic and thus extremely painful. As much as 80% of ulcers are associated with Helicobacter pylori, a spiral-shaped bacterium that lives in the acidic environment of the stomach, however only 20% of those cases go to a doctor. Ulcers can also be caused or worsened by drugs such as Aspirin and other NSAIDs. Contrary to general belief, more peptic ulcers arise in the duodenum (first part of the small intestine, just after the stomach) than in the stomach. About 4% of stomach ulcers are caused by a malignant tumor, so multiple biopsies are needed to make sure. Duodenal ulcers are generally benign.

Tobacco Smoking, not eating properly, blood group, spices and other factors that were suspected to cause ulcers until mokinglate in the 20th century, are actually of relatively minor importance in the development of peptic ulcers.

A major causative factor (60% of gastric and up to 90% of duodenal ulcers) is chronic inflammation due to Helicobacter pylori that colonizes (i.e. settles there after entering the body) the antral mucosa. The immune system is unable to clear the infection, despite the appearance of antibodies. Thus, the bacterium can cause a chronic active gastritis (type B gastritis), resulting in a defect in the regulation of gastrin production by that part of the stomach, and gastrin secretion can either be decreased (most cases) resulting in hypo- or achlorhydria or increased. Gastrin stimulates the production of gastric acid by parietal cell and, in H. pylori colonization responses that increase gastrin, the increase in acid can contribute to the erosion of the mucosa and therefore ulcer formation. Studies have shown eating cabbage or cabbage juice can increase the mucosa lining in the stomach.

Another major cause is the use of NSAIDs (see above). The gastric mucosa protects itself from gastric acid with a layer of mucus, the secretion of which is stimulated by certain prostaglandins. NSAIDs block the function of cyclooxygenase 1 (cox-1), which is essential for the production of these prostaglandins. Newer NSAIDs (celecoxib, rofecoxib) only inhibit cox-2, which is less essential in the gastric mucosa, and roughly halve the risk of NSAID-related gastric ulceration. As the prevalence of H. pylori-caused ulceration declines in the Western world due to increased medical treatment, a greater proportion of ulcers will be due to increasing NSAID use among individuals with pain syndromes as well as the growth of aging populations that develop arthritis.

Glucocorticoids lead to atrophy of all epithelial tissues. Their role in ulcerogenesis is relatively small.

There is debate as to whether Stress in the psychological sense can influence the development of peptic ulcers (see Stress and ulcers). Burns and head trauma, however, can lead to "stress ulcers", and it is reported in many patients who are on mechanical ventilation.

Smoking leads to atherosclerosis and vascular spasms, causing vascular insufficiency and promoting the development of ulcers through ischemia.

Overuse of laxatives is also known to cause peptic ulcers.

A family history is often present in duodenal ulcers, especially when blood group O is also present. Inheritance appears to be unimportant in gastric ulcers.

Gastrinomas (Zollinger Ellison syndrome), rare gastrin-secreting tumors, cause multiple and difficult to heal ulcers.


 

 

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